By Icon Health Publications
This can be a 3-in-1 reference e-book. It provides a whole clinical dictionary overlaying hundreds of thousands of phrases and expressions when it comes to glutamine. It additionally provides wide lists of bibliographic citations. ultimately, it offers details to clients on tips to replace their wisdom utilizing a variety of web assets. The ebook is designed for physicians, clinical scholars getting ready for Board examinations, clinical researchers, and sufferers who are looking to familiarize yourself with study devoted to glutamine. in case your time is efficacious, this e-book is for you. First, you won't waste time looking the web whereas lacking loads of suitable info. moment, the booklet additionally saves you time indexing and defining entries. eventually, you won't waste money and time printing countless numbers of web content.
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This can be a 3-in-1 reference booklet. It provides an entire clinical dictionary protecting thousands of phrases and expressions when it comes to glutamine. It additionally supplies wide lists of bibliographic citations. ultimately, it presents details to clients on the way to replace their wisdom utilizing a variety of web assets.
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Additional info for Glutamine - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References
This will be achieved by the following steps: (1) Isolating peptides that bindhsGFA1 using a modified yeast two hybrid assay. (2) Determine which peptides inhibit hsGFA1 activity by examining the conditional lethality of gfa1-/pGaI-hsGFA1+ yeast, that are viable despite to loss of the endogenous gfa1 gene due to rescue by galactose- regulated expression of hsGFA1. Inhibitors of hsGFA1 in these yeast cells will be cytotoxic. (3) Expressing the hsGFA1 peptide inhibitors in insulin-responsive cultured HepG2 hepatic cells and3T3-L1 adipocytes and ascertaining effects on GFA activity and the hexosamine pathway, as well as cellular glucose transport.
Clinical/pre-clinical data suggest OXP-5FU synergy; mechanisms behind the synergy are not understood. Prior studies have shown that response and survival after therapy with 5FU (colon cancer) or 5FU/cisplatin (gastric cancer) are inversely associated with thymidylate synthase (TS), dihydropyrmidine dehydrogenase (DPD) and the excision repair cross-complementing-1 (ERCC-1) gene expressions. Studies of OXP resistant cell lines indicate that resistance is multifactorial, as evidenced by lowered drug accumulation, increased glutathione and decreased DNA-Pt adducts.
The applicant's previous work in characterizing the cellular physiology of the newly discovered neuropeptide hypocretin/orexin is the first step in his journey to pursue his long-term goal of understanding neurotransmission in the LH. The funding of this research proposal is important for the applicant to continue this course. A career development plan is also documented in this proposal. Generate_Screen • Project Title: MECHANISM CYTOTOXICITY OF ANDROGEN RECEPTOR INDUCED Principal Investigator & Institution: Lafevre-Bernt, Michelle; Buck Institute for Age Research Novato, Ca 94945 Timing: Fiscal Year 2003; Project Start 01-AUG-2003; Project End 31-JUL-2005 Summary: (provided by applicant): Kennedy's disease, or spinal and bulbar muscular atrophy (SBMA) is caused by an expansion of CAG repeats (which encode for glutamine) in the 5' end of the coding region of the androgen receptor (AR) gene.
Glutamine - A Medical Dictionary, Bibliography, and Annotated Research Guide to Internet References by Icon Health Publications