By V. Bernard, William
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Extra info for Equine Pediatric Medicine
Unfortunately, neonatal foals appear to compensate poorly for hypovolemia, which not only makes the effects more severe than in mature horses, but also makes hypovolemia harder to identify. In mature horses the signs of hypovolemia are tachycardia, cold extremities, tachypnea, decreased pulse pressure, and reduced jugular pulse. Unfortunately, none of these signs are consistently present in hypovolemic foals. In a series of referred cases, 20 foals had a mean arterial pressure of <60 mmHg on admission.
Severe hypomagnesemia in the horse can result in ventricular arrhythmias and also muscle tremors, ataxia, seizures, and calcification of elastic tissue. Other clinical manifestations of hypomagnesemia reported in humans include supraventricular tachycardia, atrial fibrillation, thrombosis, anemia, decreased muscle strength, increased nephrotoxicity of aminoglycoside drugs, increased pulmonary vascular resistance, and sudden death. Hypomagnesemia can also result in hypokalemia refractory to potassium supplementation.
These findings may suggest that norepinephrine, which is also an agonist for beta-1 receptors and, therefore, a positive inotrope, may have advantages over phenylephrine as a vasopressor. If phenylephrine is used as a vasopressor, it should be used in conjunction with a beta agonist such as dobutamine. Dopamine Dopamine is an alpha-, beta-1-, and beta-2-adrenergic receptor agonist and an agonist for dopaminergic receptors. This results in a complicated drug profile, where different effects predominate at different doses.
Equine Pediatric Medicine by V. Bernard, William