Read e-book online B Chromosomes In The Eukaryote Genome (Cytogenetic & Genome PDF

By J.P.M. Camacho

ISBN-10: 3805578075

ISBN-13: 9783805578073

B chromosomes are extra passengers present in the karyotypes of approximately 15 consistent with cent of eukaryote species. they're most sensible understood as genome parasites exploiting the host genome due to their transmissional virtue, and are often deleterious for the organism sporting them. because the final compilation of B chromosome literature greater than two decades in the past, many new findings have contributed to development an expanding physique of data of those mysterious genetic components. This unmarried subject factor of "Cytogenetic and Genome study" updates those new information in its forty papers authored via researchers from 19 nations. It offers updated stories on vegetation, fish, amphibians and mammals and, for the 1st time, on Sternorrhyncha hemipterans, decapodan crustaceans, helminth parasites and lizards. It additionally provides new unique information on many points of B chromosomes together with their definition, new occurrences, frequency, constitution, composition, foundation, results at the host genome, and extra.

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Extra info for B Chromosomes In The Eukaryote Genome (Cytogenetic & Genome Research)

Sample text

C . . C . . C . TGC . . . . . . . . . . . . . . . . . . . . . . . . . . . . . Dashes represent deletions, and boxes denote polymorphisms in exon regions each locus (Fig. 2). Consensus sequences were generated from the putative non-B alleles (sequences from group 1 and 2; Fig. 2) and hypothetical aneuploid alleles (group 3; Fig. 2), and comparisons of restriction enzyme sites between these sequences yielded diagnostic cleavage sites (Fig. 3a). For locus MVI11, an AlwNI site was found at approximately position 150 on the putative aneuploid allele while no corresponding site was found on the non-B allele (Fig.

TGAC . A . . . T . . . TGAC . A . . . T . . . TGAC . A . . . T . . . TGAC . A . G . . T . . . T . A . CAC . . . T . . . T . A . A . A . . T . . . T . A . A . . . T . . . T . A . A . . . T . . . T . A . A . . . T . . . T . A . A . . . T TAAGGATC . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . .

1995). , 2002). , 1998). , 1998). Here again, the predominant maternal origin is not related to accumulation. In Type III HSCs, the estimated risk of producing malformations is 28 %, with the exception of isochromosomes 12 and 18 that always result in a severely affected phenotype (Crolla, 1998). As a rule, the risk of producing malformations for any non-acrocentric HSC is directly related to the size of the original chromosome. , 1995). Y chromosome markers may also be associated to phenotypic defects.

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B Chromosomes In The Eukaryote Genome (Cytogenetic & Genome Research) by J.P.M. Camacho


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